Search results for "IgA Deficiency"

showing 8 items of 8 documents

Celiac disease and selective immunoglobulin A deficiency

1997

Selective IgA deficiency was observed in 12 of 688 (1.7%) patients with celiac disease who were clinically undistinguishable from patients with celiac disease with normal IgA levels. This high prevalence of IgA deficiency in patients with celiac disease makes serum IgA assay advisable when screening for celiac disease is performed by measurement of antigliadin antibodies or anti-IgA endomysium antibodies. Similarly, subjects with IgA deficiency should be considered at risk of celiac disease.

AdolescentGlutensCross-sectional studyMuscle Fibers SkeletalDiseaseSelective IgA deficiencyImmunoglobulin EGliadinCoeliac diseaseMyofibrilsRisk FactorsImmunopathologyConfidence IntervalsDiet Protein-RestrictedPrevalencemedicineHumansChildChi-Square Distributionbiologybusiness.industryAge FactorsIgA DeficiencyInfantnutritional and metabolic diseasesmedicine.diseaseEndomysiumdigestive system diseasesImmunoglobulin ACeliac DiseaseIntestinal DiseasesCross-Sectional Studiesmedicine.anatomical_structureImmunoglobulin MChild PreschoolImmunoglobulin GPediatrics Perinatology and Child HealthImmunologybiology.proteinAntibodybusinessFollow-Up StudiesThe Journal of Pediatrics
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Cytokine genotyping (TNF and IL-10) in patients with celiac disease and selective IgA deficiency

2003

Selective IgA deficiency (IgAD) and celiac disease (CD) are frequently associated and share the ancestral haplotype human leukocyte antigen (HLA)-8.1, which is characterized by a peculiar cytokine profile. The aim of this study was to evaluate the role of tumor necrosis factor (TNF) and interleukin (IL)-10 alleles in CD and CD-IgAD.The distribution of some biallelic polymorphisms of both cytokine promoters (-308G--A and -863C--A at TNF promoter sequence and -1082G--A, -819C--A, and -592C--T at IL-10 promoter) were typed using biotilinated specific probes in 32 celiac patients, in 34 CD-IgAD patients, and in 96 healthy controls.In CD and CD-IgAD, the -308A allele was significantly more frequ…

AdultGenotypemedicine.medical_treatmentDiseaseSelective IgA deficiencyCoeliac diseaseGene FrequencyImmunopathologyHumansMedicineChildPromoter Regions GeneticGenotypingPolymorphism GeneticHepatologyTumor Necrosis Factor-alphabusiness.industryIgA DeficiencyGastroenterologynutritional and metabolic diseasesmedicine.diseasedigestive system diseasesInterleukin-10Celiac DiseaseInterleukin 10Cross-Sectional StudiesCytokineImmunologyCytokinesTumor necrosis factor alphabusinessSequence AnalysisThe American Journal of Gastroenterology
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Anaphylaktischer Schock nach Lipid-Austauschchromatographie bei einer Patientin mit angeborenem IgA-Mangel

1995

We report on a 59-year-old woman patient with selective IgA deficiency and familial hypercholesterinaemia. To lower elevated LDL levels in the patient's blood, immunoadsorption therapy with sheep-anti-human polyclonal antibodies coupled to sepharose columns was administered. During the procedure, the patient developed an anaphylactic shock requiring intensive care treatment. The patient's history revealed a fresh cell therapy with fetal sheep cells 10 years previously. Intracutaneous testing confirmed sensitization to sheep immunoglobulin, which was the most likely reason for the anaphylactic shock.

Allergybiologybusiness.industryDermatologySelective IgA deficiencyImmunoglobulin Emedicine.diseasemedicine.anatomical_structureImmunopathologyShock (circulatory)Immunologybiology.proteinmedicinemedicine.symptomAntibodyImmunoadsorptionbusinessSensitizationDer Hautarzt
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Dermatitis ulcerosa und ?A-Mangel

1970

Der Zusammenhang der Dermatitis ulcerosa mit Storungen des Immunglobulinstoffwechsels ist evident. Bei einer Patientin war erstmals ein IgA-Mangel bei Dermatitis ulcerosa nachweisbar, wahrend bisher nur Paraproteinamien gefunden wurden. Auf die Bedeutung dieses Befundes und die diesbezugliche Literatur wird eingegangen.

Gynecologymedicine.medical_specialtybusiness.industryMedicineIgA deficiencyDermatologyGeneral MedicinebusinessArchiv f�r Klinische und Experimentelle Dermatologie
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Compensatory IgM to the Rescue: Patients with Selective IgA Deficiency Have Increased Natural IgM Antibodies to MAA-LDL and No Changes in Oral Microb…

2021

Abstract IgA is the most abundant Ab in the human body. However, most patients with selective IgA deficiency (SIgAD) are asymptomatic. IgM, and to lesser extent IgG Abs, are generally presumed to compensate for the lack of IgA in SIgAD by multiplying and adopting functions of IgA. We used data from the Northern Finland Birth Cohort 1966 to investigate whether SIgAD patients have differences in levels of natural Abs to oxidized epitopes compared with 20 randomly selected healthy controls. First, we screened the saliva and serum samples from the Northern Finland Birth Cohort 1966 cohort (n = 1610) for IgA concentration. We detected five IgA-deficient subjects, yielding a prevalence of 0.3%, w…

MaleSalivaImmunologySelective IgA deficiencyGut floraAsymptomaticEpitopesuuimmunologiaMalondialdehydeRNA Ribosomal 16SmedicineImmunology and AllergyHumanslimakalvotSalivaFinlandimmuunivajausoireyhtymätbiologyBacteriabusiness.industryvasta-aineetIgA DeficiencyGeneral MedicineMiddle Agedmedicine.diseasebiology.organism_classificationGastrointestinal MicrobiomeImmunoglobulin ALipoproteins LDLmikrobistoImmunoglobulin MCase-Control StudiesImmunoglobulin GImmunologyCohortBirth CohortFemale3111 Biomedicinemedicine.symptombusinessDysbiosisLipoproteinImmunoHorizons
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Pili Annulati Coincident with Alopecia Areata, Autoimmune Thyroid Disease, and Primary IgA Deficiency: Case Report and Considerations on the Literatu…

2012

Pili annulati is a rare autosomal dominant hair disorder clinically characterized by a pattern of alternating bright and dark bands of the hair, the bright bands appearing dark if observed by transmitted light. This pattern is due to the periodic occurrence of air-filled cavities along the hair cortex which scatter and reflect the light while precluding its transmission. A susceptibility region, including a possibly responsible Frizzled gene, has been mapped to the telomeric region of chromosome 12q, although a specific mutation has not been identified. The condition has sometimes been observed in concurrence with alopecia areata, and in this paper we report a case in whom the concomitant s…

Pathologymedicine.medical_specialtyAlopecia areataAutoimmune thyroid diseaseDermatologyPili annulati · Alopecia areata · Molecular changes · Autoimmune diseaseImmune systemAutoimmune diseaselcsh:DermatologymedicineSettore MED/35 - Malattie Cutanee E VenereeIgA deficiencyskin and connective tissue diseasesAutoimmune diseaseintegumentary systembusiness.industryPublished online: November 2012Molecular changeslcsh:RL1-803Alopecia areatamedicine.diseasePili annulatiHair rootHair DisorderImmunologysense organsbusinessPili annulatiCase Reports in Dermatology
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IgG1 antiendomysium and IgG antitissue transglutaminase (anti-tTG) antibodies in coeliac patients with selective IgA deficiency

2000

Background—In selective IgA deficiency (IgAD), there is no reliable screening test for coeliac disease (CD). Aim—To evaluate the usefulness of IgG1 antiendomysium and IgG antitissue transglutaminase tests for CD diagnosis in IgAD. Methods—IgA and IgG antigliadin antibodies (IgA- and IgG-AGA), IgA and IgG1 antiendomysium antibodies (IgA- and IgG1-EMA), and IgA and IgG antitissue

Screening testbiologyTissue transglutaminaseGastroenterologySelective IgA deficiencymedicine.diseaseEndomysiumCoeliac diseaseAntiendomysium antibodiesmedicine.anatomical_structureImmunopathologyImmunologymedicinebiology.proteinAntibodyGut
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Plasma cytokine profiles in patients with celiac disease and selective IgA deficiency

2003

Celiac disease (CD) and selective IgA deficiency (IgAD) are frequently associated, and share the same genetic background. The aim of the present study was to evaluate both Type 1 and 2 plasma cytokine levels in CD and in CD-IgAD. IL-2, TNF-alpha, IL-10, IL-4 and IL-13 plasma levels were measured both at diagnosis and after a gluten-free diet (GFD) in 32 CD patients, in 27 CD-IgAD patients and in 30 healthy controls. IFN-gamma levels were significantly higher in CD and CD-IgAD than in controls, TNF-alpha displayed significantly higher levels in CD-IgAD when compared both with controls and with CD, and IL-2 was in CD-IgAD significantly increased respect to controls. Kinetics of the Type 1 cyt…

business.industrymedicine.medical_treatmentImmunologyDiseasePlasma levelsSelective IgA deficiencymedicine.diseaseCoeliac diseaseCytokineImmunopathologyPediatrics Perinatology and Child HealthImmunologyBlood plasmamedicineImmunology and AllergyIn patientbusinessPediatric Allergy and Immunology
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